<?xml version="1.0" encoding="UTF-8"?><xml><records><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Jessica Rauch</style></author><author><style face="normal" font="default" size="100%">Philip Eisermann</style></author><author><style face="normal" font="default" size="100%">Bernd Noack</style></author><author><style face="normal" font="default" size="100%">Ute Mehlhoop</style></author><author><style face="normal" font="default" size="100%">Birgit Muntau</style></author><author><style face="normal" font="default" size="100%">Johannes Schäfer</style></author><author><style face="normal" font="default" size="100%">Dennis Tappe</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Typhus Group Rickettsiosis, Germany, 2010–20171</style></title><secondary-title><style face="normal" font="default" size="100%">Emerging Infectious Diseases</style></secondary-title><short-title><style face="normal" font="default" size="100%">Emerg. Infect. Dis.</style></short-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Germany</style></keyword><keyword><style  face="normal" font="default" size="100%">lice</style></keyword><keyword><style  face="normal" font="default" size="100%">rickettsia prowazekii</style></keyword><keyword><style  face="normal" font="default" size="100%">Rickettsia typhi</style></keyword><keyword><style  face="normal" font="default" size="100%">Rickettsiosis</style></keyword><keyword><style  face="normal" font="default" size="100%">typhus</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">2018</style></year><pub-dates><date><style  face="normal" font="default" size="100%">July 2018</style></date></pub-dates></dates><urls><web-urls><url><style face="normal" font="default" size="100%">http://wwwnc.cdc.gov/eid/article/24/7/18-0093_article.htm</style></url></web-urls></urls><volume><style face="normal" font="default" size="100%">24</style></volume><pages><style face="normal" font="default" size="100%">1213 - 1220</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p&gt;Typhus group rickettsiosis is caused by the vectorborne bacteria Rickettsia typhi and R. prowazekii. R. typhi, which causes murine typhus, the less severe endemic form of typhus, is transmitted by fleas; R. prowazekii, which causes the severe epidemic form of typhus, is transmitted by body lice. To examine the immunology of human infection with typhus group rickettsiae, we retrospectively reviewed clinical signs and symptoms, laboratory changes, and travel destinations of 28 patients who had typhus group rickettsiosis diagnosed by the German Reference Center for Tropical Pathogens, Hamburg, Germany, during 2010&amp;ndash;2017. Immunofluorescence assays of follow-up serum samples indicated simultaneous seroconversion of IgM, IgA, and IgG or concurrence in the first serum sample. Cytokine levels peaked during the second week of infection, coinciding with organ dysfunction and seroconversion. For 3 patients, R. typhi was detected by species-specific nested quantitative PCR. For all 28 patients, R. typhi was the most likely causative pathogen.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">7</style></issue></record></records></xml>